Results: 1-15 |
Results: 15

Authors: Heyworth, PG Curnutte, JT Rae, J Noack, D Roos, D van Koppen, E Cross, AR
Citation: Pg. Heyworth et al., Hematologically important mutations: X-linked chronic granulomatous disease (Second update), BL CELL M D, 27(1), 2001, pp. 16-26

Authors: Rae, J
Citation: J. Rae, Organizing participation in interaction: Doing participation framework, RES LANG S, 34(2), 2001, pp. 253-278

Authors: Green, SP Cairns, B Rae, J Errett-Baroncini, C Hongo, JAS Erickson, RW Curnutte, JT
Citation: Sp. Green et al., Induction of gp91-phox, a component of the phagocyte NADPH oxidase, in microglial cells during central nervous system inflammation, J CEREBR B, 21(4), 2001, pp. 374-384

Authors: Muench, MO Rae, J Barcena, A Leemhuis, T Farrell, J Humeau, L Maxwell-Wiggins, JR Capper, J Mychaliska, GB Albanese, CT Martin, T Tsukamoto, A Curnutte, JT Harrison, MR
Citation: Mo. Muench et al., Transplantation of a fetus with paternal Thy-1(+)CD34(+) cells for chronicgranulomatous disease, BONE MAR TR, 27(4), 2001, pp. 355-364

Authors: Shields, RL Namenuk, AK Hong, K Meng, YG Rae, J Briggs, J Xie, D Lai, J Stadlen, A Li, B Fox, JA Presta, LG
Citation: Rl. Shields et al., High resolution mapping of the binding site on human IgG1 for Fc gamma RI,Fc gamma RII, Fc gamma RIII, and FcRn and design of IgG1 variants with improved binding to the Fc gamma R, J BIOL CHEM, 276(9), 2001, pp. 6591-6604

Authors: Noack, D Rae, J Cross, AR Ellis, BA Newburger, PE Curnutte, JT Heyworth, PG
Citation: D. Noack et al., Autosomal recessive chronic granulomatous disease caused by defects in NCF-1, the gene encoding the phagocyte p47-phox: mutations not arising in the NCF-1 pseudogenes, BLOOD, 97(1), 2001, pp. 305-311

Authors: Cross, AR Noack, D Rae, J Curnutte, JT Heyworth, PG
Citation: Ar. Cross et al., Hematologically important mutations: The autosomal recessive forms of chronic granulomatous disease (First update), BL CELL M D, 26(5), 2000, pp. 561

Authors: Rae, J Noack, D Heyworth, PG Ellis, BA Curnutte, JT Cross, AR
Citation: J. Rae et al., Molecular analysis of 9 new families with chronic granulomatous disease caused by mutations in CYBA, the gene encoding p22(phox), BLOOD, 96(3), 2000, pp. 1106-1112

Authors: Roesler, J Curnutte, JT Rae, J Barrett, D Patino, P Chanock, SJ Goerlach, A
Citation: J. Roesler et al., Recombination events between the p47-phox gene and its highly homologous pseudogenes are the main cause of autosomal recessive chronic granulomatous disease, BLOOD, 95(6), 2000, pp. 2150-2156

Authors: Kavanaugh, AF Wolfe, F Auslander, M Bresloff, T Clark, DL Hillman, D Lewis, DL Martinez, L Milgram, L Newcomer, D Nguyen, T Rae, J
Citation: Af. Kavanaugh et al., Roundtable II: Cost concerns for rheumatoid arthritis treatment, AM J M CARE, 5(14), 1999, pp. S880-S888

Authors: Noack, D Heyworth, PG Curnutte, JT Rae, J Cross, AR
Citation: D. Noack et al., A novel mutation in the CYBB gene resulting in an unexpected pattern of exon skipping and chronic granulomatous disease, BBA-MOL BAS, 1454(3), 1999, pp. 270-274

Authors: Noack, D Rae, J Cross, AR Munoz, J Salmen, S Mendoza, JA Rossi, N Curnutte, JT Heyworth, PG
Citation: D. Noack et al., Autosomal recessive chronic granulomatous disease caused by novel mutations in NCF-2, the gene encoding the p67-phox component of phagocyte NADPH oxidase, HUM GENET, 105(5), 1999, pp. 460-467

Authors: Patino, PJ Rae, J Noack, D Erickson, R Ding, JB de Olarte, DG Curnutte, JT
Citation: Pj. Patino et al., Molecular characterization of autosomal recessive chronic granulomatous disease caused by a defect of the nicotinamide adenine dinucleotide phosphate(reduced form) oxidase component p67-phox, BLOOD, 94(7), 1999, pp. 2505-2514

Authors: Kerby, J Rae, J
Citation: J. Kerby et J. Rae, Moral identity in action: Young offenders' reports of encounters with the police, BR J SOC P, 37, 1998, pp. 439-456

Authors: Foster, CB Lehrnbecher, T Mol, F Steinberg, SM Venzon, DJ Walsh, TJ Noack, D Rae, J Winkelstein, JA Curnutte, JT Chanock, SJ
Citation: Cb. Foster et al., Host defense molecule polymorphisms influence the risk for immune-mediatedcomplications in chronic granulomatous disease, J CLIN INV, 102(12), 1998, pp. 2146-2155
Risultati: 1-15 |