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Citation: Cm. Fraser et Mr. Dando, Genomics and future biological weapons: The need for preventive action by the biomedical community, NAT GENET, 29(3), 2001, pp. 253-256
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Citation: Rb. Dyer et Ct. Mcmurray, Mutant protein in Huntington disease is resistant to proteolysis in affected brain, NAT GENET, 29(3), 2001, pp. 270-278
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Citation: Tk. Kawaguchi et al., The AZFc region of the Y chromosome features massive palindromes and uniform recurrent deletions in infertile men, NAT GENET, 29(3), 2001, pp. 279-286
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Citation: A. Trockenbacher et al., MID1, mutated in Opitz syndrome, encodes an ubiquitin ligase that targets phosphatase 2A for degradation, NAT GENET, 29(3), 2001, pp. 287-294
Citation: Tg. Graeber et D. Eisenberg, Bioinformatic identification of potential autocrine signaling loops in cancers from gene expression profiles, NAT GENET, 29(3), 2001, pp. 295-300
Citation: Hm. Hoffman et al., Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle-Wells syndrome, NAT GENET, 29(3), 2001, pp. 301-305
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Citation: R. Birkenhager et al., Mutation of BSND causes Bartter syndrome with sensorineural deafness and kidney failure, NAT GENET, 29(3), 2001, pp. 310-314
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Citation: M. Nakano et al., Homozygous mutations in ARIX (PHOX2A) result in congenital fibrosis of theextraocular muscles type 2, NAT GENET, 29(3), 2001, pp. 315-320